Nuclear interaction between ADR-induced p65 and p53 mediates cardiac injury in iNOS (-/-) mice.

Elevated glutathione is not sufficient to protect against doxorubicin-induced nuclear damage in heart in multidrug resistance-associated protein 1 (Mrp1/Abcc1) null mice.

Elevated glutathione is not sufficient to protect against doxorubicin-induced nuclear damage in heart in multidrug resistance-associated protein 1 (Mrp1/Abcc1) null mice.

[Anonymous].  2015.  Elevated glutathione is not sufficient to protect against doxorubicin-induced nuclear damage in heart in multidrug resistance-associated protein 1 (Mrp1/Abcc1) null mice.. The Journal of pharmacology and experimental therapeutics. 355(2):272-9.

Novel role of 4-hydroxy-2-nonenal in AIFm2-mediated mitochondrial stress signaling.

RelB Expression Determines the Differential Effects of Ascorbic Acid in Normal and Cancer Cells.

Inhibition of the integrin/FAK signaling axis and c-Myc synergistically disrupts ovarian cancer malignancy.

A novel redox regulator, MnTnBuOE-2-PyP<sup>5+</sup>, enhances normal hematopoietic stem/progenitor cell function.

Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury.

Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury.

[Anonymous].  2018.  Extracellular Vesicles Released by Cardiomyocytes in a Doxorubicin-Induced Cardiac Injury Mouse Model Contain Protein Biomarkers of Early Cardiac Injury.. Clinical cancer research : an official journal of the American Association for Cancer Research. 24(7):1644-1653.

Redox Paradox: A Novel Approach to Therapeutics-Resistant Cancer.

Extracellular redox state shift: A novel approach to target prostate cancer invasion.

Hepatitis C treatment in an urban population.