Methamphetamine induces AP-1 and NF-kappaB binding and transactivation in human brain endothelial cells.

Unsaturated fatty acids selectively induce an inflammatory environment in human endothelial cells.

PCB-induced oxidative stress in endothelial cells: modulation by nutrients.

Linoleic acid induces MCP-1 gene expression in human microvascular endothelial cells through an oxidative mechanism.

Proinflammatory properties of coplanar PCBs: in vitro and in vivo evidence.

Linoleic acid-induced VCAM-1 expression in human microvascular endothelial cells is mediated by the NF-kappa B-dependent pathway.

Measurement of inflammatory properties of fatty acids in human endothelial cells.

Methamphetamine-induced TNF-alpha gene expression and activation of AP-1 in discrete regions of mouse brain: potential role of reactive oxygen intermediates and lipid peroxidation.

Methamphetamine-induced TNF-alpha gene expression and activation of AP-1 in discrete regions of mouse brain: potential role of reactive oxygen intermediates and lipid peroxidation.

[Anonymous].  2002.  Methamphetamine-induced TNF-alpha gene expression and activation of AP-1 in discrete regions of mouse brain: potential role of reactive oxygen intermediates and lipid peroxidation.. Neuromolecular medicine. 2(1):71-85.

Methamphetamine activates DNA binding of specific redox-responsive transcription factors in mouse brain.

Redox-regulated mechanisms of IL-4-induced MCP-1 expression in human vascular endothelial cells.