Endovascular management of blunt traumatic renal artery dissection.

Phospholemman-mediated activation of Na/K-ATPase limits Na i and inotropic state during beta-adrenergic stimulation in mouse ventricular myocytes.

Phospholemman-mediated activation of Na/K-ATPase limits Na i and inotropic state during beta-adrenergic stimulation in mouse ventricular myocytes.

[Anonymous].  2008.  Phospholemman-mediated activation of Na/K-ATPase limits Na i and inotropic state during beta-adrenergic stimulation in mouse ventricular myocytes.. Circulation. 117(14):1849-55.

Stent-graft migration following endovascular repair of aneurysms with large proximal necks: anatomical risk factors and long-term sequelae.

Stent-graft migration following endovascular repair of aneurysms with large proximal necks: anatomical risk factors and long-term sequelae.

[Anonymous].  2002.  Stent-graft migration following endovascular repair of aneurysms with large proximal necks: anatomical risk factors and long-term sequelae.. Journal of endovascular therapy : an official journal of the International Society of Endovascular Specialists. 9(5):652-64.

Na+ transport in cardiac myocytes; Implications for excitation-contraction coupling.

Exactly 250 feet.

Extracellular potassium dependence of the Na+-K+-ATPase in cardiac myocytes: isoform specificity and effect of phospholemman.

Extracellular potassium dependence of the Na+-K+-ATPase in cardiac myocytes: isoform specificity and effect of phospholemman.

[Anonymous].  2009.  Extracellular potassium dependence of the Na+-K+-ATPase in cardiac myocytes: isoform specificity and effect of phospholemman.. American journal of physiology. Cell physiology. 297(3):C699-705.

Accuracy of three-dimensional simulation in the sizing of aortic endoluminal devices.

Isoform specificity of the Na/K-ATPase association and regulation by phospholemman.

A risk index for sternal wound infection after cardiovascular surgery.

Na/K-ATPase--an integral player in the adrenergic fight-or-flight response.